B. Chloroquine

C. Chlorpheniramine

D. Chlorhexidine

A. Olanzepine

C. Risperidone

D. Clozapine

A. Naltrexone

B. Furazolidone

C. Naloxone

A. They act as neuromodulators

C. They are acidic precursors of Brain amines

D. They are metabolites of various neurogenic amines

A. NSAIDS benefit by preventing prostaglandin synthesis

B. Analgesia is associated with ? and k receptors

D. Dysphoria is associated with S receptors

A. Zopiclone

B. Diazepam

D. Nitrazepam

A. Indomethacin

B. Phenyl butazone

C. Aspirin

A. Beclofen

B. Diazepam

C. Mephensin

A. Alzhiemers disease

B. Cushings syndrome

C. Vitamin C deficiency

A. Produce analgesia

C. Suppress coughing

D. Produce muscle relaxation

B. Fever

C. Post myocardial infarction

D. Venous thrombosis

B. CNS stimulation

C. CNS depression

D. Decreased salivation

A. Pheno barbital

C. Sodium nitroprusside

D. Glyceryl trinitrite

A. It produces more acetycholine receptors

B. It increases the action of cholinesterase

C. It is produces more acetylcholine

B. Stage I

C. Stage II

D. Stage III plane 2

A. Esophageal spasm

B. Cyanide poisoning

C. CCF

B. Diphenydramine

C. Promethazine

D. Pyrilamine

A. The therapeutic index of the drug

B. Range of disease in which the drug beneficial

C. The therapeutic does range of the drug

A. Sodium citrate

B. Ondansetron

D. Atropine

B. 84%

C. 80.50%

D. 4.75%

A. It causes excertion of Na & Cl

B. It causes metabolic alkalosis

C. It is very potent

A. Prothrombin

B. Acetylsalicylic acid

C. Plasmin

A. Infantile spasm

B. Grand mal epilepsy

D. None of the above

A. A normal healthy individual

C. A patient with mild systemic disease

D. None of the above

B. Nutritional component

C. Psychological component

D. Psysiological component

A. Morbidity index

C. Moratality index

D. Anesthetic ratio

A. Flunarizine

B. Propanolol

C. Divalprox sodium

A. Ibuprofen

B. Aspirin

C. Indomethacin

B. phenytoin

C. Corticosteroids

D. Diazepam

B. High K+ levels

C. High Na+ levels

D. High Ca2+ levels

A. Increase in total amount of circulatory neutrophils

C. Anti-inflammatory effect is increased

D. Inhibit phospholipase A2

A. Thiopentone

C. Nitrous oxide

D. Tentanyl

B. Inhibition of thromboxane TXA2

C. Prolonged bleeding time

D. Inhibition of prostaglandin PGI2

A. Paracetamol

C. Verapamil

D. Salbutamol

A. Td50

B. Md50

D. Ld50

A. Affinity

B. Purity of Enzyme

C. Physiological role

A. Poorly suited for oral administration

B. High membrane penetrability

D. The effect is more pronounced in venous

A. Decrease action of prothrombin III

C. Known to cause idiopathic thrombocytosis

D. Mixture of sulphated polypeptide

B. cAMP

C. cGMP

D. PGI2

A. Ciprofloxacin

C. Diphenhydramine

D. Carbamezapine

A. Adrenal steroids

B. H1 histamine receptor antagonist

C. Anticholinergic

A. Shock

C. Anaphylaxis

D. Asthma

B. Toxicity effect

C. Drug on Drug effect

D. Side effect

B. White blood cells

C. Serum

D. Haemoglobin

A. Are more common than first order kinetics

B. Show a plot of drug concentration versus time that is linear

D. Decrease in concentration exponentially with time

B. Cerebral blood flow

C. Partial pressure of GA

D. Blood solubility

B. Half life

C. Clearance

D. Rate of administration

A. Atosiban

B. Ritodrine

D. Isoxsuprine

B. Use in head injury patient

C. Use of agonist with mixed agonist-antagonist

D. use in impaired pulmonary function

A. Carcinogens

C. All of the above

D. Allergens