A. Biclonal in origin

C. None of the above

D. Multiclonal in origin

A. Regeneration

C. Cicatrisation

D. Primary healing

A. Arteries

C. Vein

D. Valve cusp

A. Lactic Dehydrogenase (LDH)

B. Creatine phospho kinase (CPK)

D. Serum glutamic oxaloacetic transaminase (SGOT)

A. Aplasia

C. Anaplasia

D. Abnormal mitosis

A. Dysplasia

C. Aplasia

D. Anaplasia

A. Blood pressure

B. Cardiac output

C. CVP to 8 cm of water

A. Epithelial changes

B. Acute inflammatory response

C. Organization

A. Tyhoid

B. Amoebiasis

C. Viral infections

A. Dopaminergic receptors

C. Opioid receptors

D. Beta 2 receptors

A. Total lack of neutrophils

B. Total lack of platelets

D. Reduced score of acid phosphatase in granulocytes

B. C2, C3

C. C3b, C5a

D. C3b, C5b

A. Ewings sarcoma

B. Giant cell tumour

C. Chondrosarcoma

A. Uterus

B. Thyroid

D. Kidney

B. Portal vein

C. Pulmonary vein

D. Hepatic vein

A. Mydriasis

B. Vasoconstriction

D. Contraction of urinary spinctors

A. Endothelial injury

B. Stasis of blood

C. Turbulence of blood

A. 20% blood

C. 30% blood

D. 10% blood

A. Adequate oxygen supply

B. Protein

D. Vitamin C

A. Chondrosarcoma

B. Osteoma

C. None of the above

A. Aplastic anemia

B. Viral infection

D. Typhoid fever

A. Diminished generation of adenosine triphosphate (ATP)

B. Detachment of ribosomes from the granular endoplasmic reticulum

D. Formation of blebs in the plasma membrane

A. Induces cell mediated immunity

B. None of the above

D. Induces immunity by activation of cytoxic T-cells

A. Increased vascular permeability

B. Stasis of blood

C. Vasodilation

A. Kidney

C. Liver

D. Lung

B. Kidneys

C. Heart

D. Intestines

B. Hamartoma

C. Plemorphic tumours

D. Choristoma

A. Atelexis

B. Achalasia

D. Cacoguesia

A. petechiae small pin point hemorrhages

B. Ecchymosis large extravasation of blood in to the skin and mucous membrane

C. Purpura small areas (1cm) of hemorrhage in to the skin and mucous membrane

B. 20 to 30% haemoglobins

C. 10 to 20% haemoglobins

D. 50 to 60% haemoglobins

A. Systemic manifestations

C. Reed Sternberg cells

D. Occurence in young adults

A. Intestine

B. Ovary

D. Lung

A. Cicatrisation

B. Secondary intention

C. All of the above

A. None

B. Karyorhexis

C. Pyknosis

A. Turner syndrome

B. Down syndrome

D. Marfan syndrome

A. Kupffer cells in liver

B. Histiocytes in connective tissues

D. Microglial cells in nervous system

A. Peripheral vasodilation

C. Increased vascular permeability

D. Endothelial injury

A. It has fibrin precipitates

B. It is associated with many types of severe inflammation

D. It induces connective tissue organization

A. Type II hyp

C. Type IV hyp

D. Type III hyp

A. Macrophages

B. Dendritic cells

C. Langerhans cells

B. Hyper chromatic and pleomorphism of tumour cells

C. All of the above

D. Neoplastic cells are cohesive

A. Klutischky cells

B. Brush cells

C. Clara cells

A. Sickle cell anaemia

B. Albinism

C. Icthyosis

A. All of the above

B. Papilloma

C. Sarcomas

A. G2 phase

B. M phase

C. S phase

B. Chemicals

C. Viruses

D. Radiation

A. None of the above

B. A transplant immunity

C. An anaphylactic shock

A. Fatty streak

B. Hypercoagulability

C. Alteration in blood flow

A. Glucose-6 phosphate dehydrogenase deficiency

B. Sickle cell trait

D. a-Thalassamia

B. Cells containing excessive melanin pigment

C. Melangocytes which are functionally more active

D. large melanocytes