A. Acromegaly
B. Rickets
C. Fibrous Dysplasia
A. Fluoride
C. Congenital syphilis
D. Rickets
A. Dentinogenesis imperfecta
B. Amelogenesis imperfecta
D. Regional odontodysplasia
A. Polysaccharide
B. Glucose
C. Dextran
A. Dextranase and soluble dextran
B. None of the above
C. soluble dextan and glucosyl transferase
A. is caused by muscle fatigue due to chronic oral habits are grinding and clenching
C. Maily affects young females
D. Treatment involves construction of occlusal guard and stress free emotional condition
A. Warts on the tongue
B. Cysts of gingiva in growing children
C. Cysts associated with soft palate
A. Mesoderm
B. Endoderm
D. Ecto and Mesoderm
A. Ectodermal dysplasia
B. Cleidocranialdysostosis
D. Papillion levefre syndrome
A. Stage III
B. Stage I
C. Stage II
A. Posterior tonsillar pillar
B. Vermilion of the lip
D. Palate
B. Degenerative arthritis
C. Traumatic arthritis
D. Trigeminal neuralgia
A. Disto molars doesnt resemble any other tooth
B. Mesiodens is the most common supernumerary tooth
D. May have resemblance to normal teeth
A. Condensing osteitis
C. Osteopetrosis
D. Fibrous dysplasia
A. Gingival cyst of newborn
C. Enamel pearls
D. Epithelial rests
B. Aphthous stomatitis
C. Oral submucous fibrosis
D. Oral lichen planus
A. Intruded teeth
B. Impacted teeth
C. Unerupted teeth
A. Lesions may be Bilaterally symmetrical on tongue
C. No treatment is required
D. May be related to emotional stress
B. Subluxation
C. None of the above
D. Dislocation
A. pain during mandibular movement
C. Stabbing type pain orginate in the tonsillar regions
D. When the jaws are closed the pain subsided
A. Down syndrome
B. Turner syndrome
C. Sturge weber syndrome
A. Type I dentinogenesis imperfecta
B. Type III dentinogenesis imperfecta
C. Dentin dysplasia
A. Frequency of sugar intake
B. Quantity of sugar intake
C. Physical form of sugar
A. Actinomyces
B. Strep. viridans
C. Lactobacillus
B. Permanent Canine
C. Permanent lateral incisor
D. Permanent second premolar
A. Herpes simplex
B. Syphilis
D. Lichen planus
B. Hyperthyroidism
C. Osteitis deformans
D. Craniofacial dysostosis
A. Primary hyperparathyroidism
B. Phantom bone disease
C. Pagets disease
A. Increase in the virulence of organisms causes infection
B. Decreased host resistance
D. Occurs in immunocompromised patients
A. Four
C. Two
D. One
B. Due to chronic suppurative abscess in over lying gingival tissue
C. Severe flurosis
D. Trauma at the time of birth
B. Division of a single tooth bud after calcification
C. Fusion of two teeth after calcification
D. Fusion of two teeth before calcification
B. Geographic tongue
C. Median rhomboid glossitis
D. Fissured tongue
B. Maxillary first premolar
C. Mandibular canine
D. Maxi. central incisor
A. Gottlieb
B. Synder
D. Miller
B. Vitamin D deficiency
C. Vitamin A deficiency
D. Teratogens
A. Dentigerous cyst OKC, radicular cyst
C. B & C
D. Dentigerous cyst, Globulomaxillary cyst, radicular cyst
A. Microdontia
B. Dens is dente
C. Anodontia
A. Loss of innervation to stapedius
B. Loss of lacrimal secretion
D. Loss of taste sensation from Ant. 2/3 of tongue
B. Osteopetrosis
C. Polyostotic fibrous dysplasia
D. Pagets disease of the bone
A. A supernumerary tooth bud enclaved within a normal tooth
C. An abnormal proliferation of pulp tissue
D. Denticle formation within the pulp tissue
A. Palate
C. Buccal mucosa
D. Lateral border of tongue
A. Hypertrophy of circumvallate papillae
B. Hypertrophy of foliate papillae
D. Hypertrophy of fungiform papillae
A. Osteopetrosis
B. Cherubism
E. Pagets disease
A. Acid phosphatase
B. Bicarbonate ion
C. Alkaline phosphatase
A. Rubinstein Taybi Syndrome
C. Mohrs syndrome
D. Sturge-Weber Syndrome
A. Aspirin administration
B. Morphine administration
C. Sublingual nitroglycerine administration
A. Maxillary premolars
C. Incisor with talon/cusp
D. Mesiodens
A. Two or three years of life
B. First month of life
C. First two years of life
A. The result of excessive fluoride ingestion
B. A hereditary disturbance
D. The result of faulty enamel matrix formation
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