B. Lungs

C. Clavicle

D. Pelvis

A. Hepatitis D

B. Hepatitis F

D. Hepatitis C

A. Haemosiderin

B. Ferritin

C. Myoglobin

B. Chronic osteomyelitis

C. Hodgkins diseaes

D. Tuberculosis

A. More sensitive, more specific

B. Less sensitive, more specific

D. Less sensitive less specific

B. Chronic infections

C. Aplastic

D. Sickle cell

A. Neoplastic change in the cell

B. Intracytoplasmic accumulation

D. Degenerative change

B. None of the above

C. HB Ag

D. HBV

A. Mycobacteria

B. Spore

D. Coccidia

A. Viral components may be synthesized but maturation & assembling is defective

B. Phase in which virus cannot be demonstrated in host cell

D. Viruses which are genetically deficient

B. HBV

C. HDV

D. HCV

B. Sickle cell anemia

C. Aplastic anemia

D. Pernicious anemia

A. Foliate deficiency anemia

B. Pernicious anemia

C. Vitamin B12 deficiency anemia

A. Subcutaneous tissue

C. Superficial skin and deep tissue

D. Systemic organs

B. Cidex

C. Ethanol

D. Boiling

B. Ascariasis and tapeworm

C. Tapeworm

D. Hood worm

A. Causes meningitis

C. Melanin production related to virulence

D. Basidiomycetes yeast

A. Lysis

B. Autopsy

C. Autolysis

A. Chronic lymphoblastic leukemia

C. Chronic monocytic leukemia

D. Chronic myeloid leukemia

A. Carcinoma

B. Ewings sarcoma

C. Melanoma

B. 24 Hrs

C. 2 weeks

D. 10 days

B. only after one year

C. antibodies are present in recurrent and chronic apthous stomatitis

D. no antibodies are present in primary HSV

A. In 70% of cases dont have any associated disease

C. In 50% of cases have some form of plasma cell dyscrasias such as multiple myeloma

D. It is more common in heart, skin and skeletal tissue

A. Pox virus

B. HIV

C. Hepatitis B virus

A. Venereal ulcer

C. Malignant pustule

D. Solar Keratosis

A. Right supraclavicular nodes

B. Left axillary nodes

D. Right axillary nodes

A. Hepatitis C virus

B. Hepatitis A virus

D. Hepatitis B virus

B. 1-2 years

C. 3-6 months

D. 1 months

A. Chocolate agar medium

B. NNN medium

C. Tellurite medium

A. Isolation form stool

C. Isolation from bile

D. Culture from blood

B. Basal cell carcinoma

C. Chronic leukemia

D. AIDS

B. Von-willebrand disease

C. Haemophilia A

D. Haemophilia B

B. Caseous necrosis

C. Liquefactive necrosis

D. Fat necrosis

A. Epithelial dysplasia

B. Neo angiogenesis

D. Liquefaction degeneration

A. Eosinophils

B. Erythrocytes

D. Monocytes

A. Scanning electron miscroscopy

B. Electron microscopy

C. X-ray crytallography

B. EACA

C. Cryoprecipitate

D. Factor VIII concentrate

A. Carrier status

B. Simple carriers

D. Late convalescence

A. Red granules

B. Brown to black granules

D. No granules

A. ELISA IS ve

B. Western Blot is ve

D. PCR is ve

A. AIDS

B. Malaria

D. Hepatitis

A. Characteristic of focal bacterial infections

C. None of the above

D. Characteristic by loss of tissue architecture

A. Antibody to HBC is not protective

C. CMI disappears soon after recover

D. Highest titres of anti HBC are found in persistent carriers of HBs Ag

B. Midline lethal granuloma

C. Sarcoidosis

D. Leprosy

B. Balantidium coli

C. Isopora belli

D. Giardia lambella

A. Skin layers

B. Normal tissues

D. Salivary glands

A. Plasmodium ovale

C. Plasmodium malariae

D. Plasmodium falciparum

A. Hepatitis C

C. Echo virus

D. HIV

A. Paradoxical carrier

B. Healthy carrier

C. Inactive carrier

A. Chromatid bars

B. Glycogen mass

C. Eccentric nucleus